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Despite these variations, the convergence of biochemical and clinical data supports a pivotal role of MI and its IPG mediators in reinforcing mitochondrial functions and mitigating OS-driven follicular dysfunction in PCOS
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2-Hydroxyestradiol (2-OHE 2 ) can experience three metabolic fates: methylation to yield 2-meOHE 2 , oxidation to form quinones, or dehydrogenation to yield 2-OHE 1
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